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If the diagnosis is confirmed, then the patient is referred for further instrumental research methods, such as: Treatment of American trypanosomiasis.
The main clinical manifestations of astelin.
Patients suffering from chronic Chagas disease are prescribed antiparasitic treatment.
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Reduces the risk of azelastine Nasal spray and regular dry and wet cleaning in homes, while not forgetting hard-to-reach places, because this is where insects hide.
The chronic phase of this disease also requires symptomatic treatment - prescribing drugs to restore the heart rhythm, installing a pacemaker, heart transplantation, and performing operations to remove the gastrostomy and colonostomy to the outer surface of the abdomen.
As mentioned earlier, living in countries such as Mexico, Central and South America, and tourist trips to these places, especially in poor areas, can lead to infection with this disease.
Unfortunately, at present there is no specific prevention of azelastine spray - suitable serums or vaccines have not yet been invented by scientists, but there are non-specific methods to prevent the risk of the disease. The spread of trypanosomiasis can be avoided by conducting health education conversations with people living in endemic areas, improving living conditions, and using correct and timely means to combat insects, in particular bed bugs. Try to avoid such places, and when in these countries, carefully monitor personal hygiene, clean skin and clothes. Pay special attention to the place of your overnight stay, because everyone knows that bedbugs are especially active at night.
People with a chronic form of American trypanosomiasis are much more difficult to treat, especially with an advanced stage of astelin, when irreversible damage has occurred in the organs of the cardiovascular system and the gastrointestinal tract.changes. With proper symptomatic treatment, a long-term remission can be achieved, but everything, again, depends on the stage of the disease and the degree of damage to organs and systems.
Usually, patients who have experienced only the acute phase of Chagas disease, without its transition to the chronic one, can hope for a full recovery, especially if the disease was diagnosed in a timely manner and the necessary treatment was started.
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Chagas disease, which occurs in the acute phase, does not affect the human body in any way in terms of subsequent complications and violations of the functionality of organs. The chronic form of the disease is much more dangerous. The vital activity of protozoan parasites, which lasts for years in human organs, leads to irreversible changes in the structure of tissues, especially muscle tissues.
Changes lead to organ fibrosis, chronic inflammation and subsequent tissue atrophy. There are chronic heart failure, a significant increase in the size of the esophagus, which leads to difficulty swallowing, expansion of the rectum, after which the normal act of defecation is disturbed. Less often, the gallbladder and its ducts, ureters undergo changes.
Chagas disease is an infection caused by a protozoan parasite (Trypanosoma cruzi) that can lead to acute inflammatory skin changes (stepomas) and can eventually cause infection and inflammation of many other tissues in the body, especially the heart and digestive tract.
Chagas disease was first described in 1909 in Brazil.
Chagas disease is caused by a protozoan parasite called Trypanosoma cruzi that is transmitted to humans from the faeces of the triatomine bug (kissing bug). can eventually reach other organs through the blood. Chagas disease can occur in three forms: acute, moderate or indefinite, and chronic.
Symptoms of Chagas disease range widely from asymptomatic to severe and chronic. Symptoms of the acute phase of Chagas disease may include swelling and/or redness at the site of skin infection (called chagas), rash, swollen lymph nodes, fever, headaches, muscle aches, fatigue, nausea, vomiting and/or diarrhea, liver and/or spleen lesions , as well as Romagna's symptom.
Symptoms and signs of the chronic phase of Chagas disease may include abnormal heart rhythms, palpitations, syncope, cardiomyopathy, congestive heart failure, shortness of breath, emphysema, stroke, sudden death, chronic abdominal pain, chronic constipation, colonic enlargement, and difficulty swallowing. Diagnosis of Chagas disease is based on the patient's medical history, physical examination, direct imaging of microscopic parasites, and detection of antibodies to parasites.
Treatment with antiparasitic drugs such as benznidazole (Rochagan, Ragonil) and nifurtimox (Lampit) kills or inhibits the development of the T. cruzi parasite.
Patients with the chronic form are usually treated with procedures to manage specific symptoms or organ damage. There is no vaccine against the parasites that cause Chagas disease in humans, but many experts believe that improved living standards and education can prevent most cases of azelastine disease.
Chagas disease (also called American trypanosomiasis) is an infection caused by a protozoan parasite (Trypanosoma cruzi) that can lead to acute inflammatory skin changes (chagomas) and eventually cause infections and inflammation in many other body tissues, especially the heart and intestinal tract.
The disease can have three phases: acute, moderate or asymptomatic, which can last from one week to two months.
Between the phases there are intermediate or indefinite stages during which the patient may experience few or no symptoms and which may last 10-20 years or more. Also, the disease may be in the chronic phase, and begins to appear after about 20 years, with more severe symptoms that develop from gradual chronic organ damage (especially the heart and intestines, although other organs may be affected), to symptoms that usually remain on all life.
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However, microscopic blood tests for the presence of astelin usually require confirmation by immunological tests because visually parasites can be mistaken for those seen in people with malaria, leishmaniasis, babesiosis, giardiasis, or African sleeping sickness. Microscopic examination and examination should be carried out by experienced specialists in the laboratory and experts in the field of parasitology.
There are several types of blood tests available to test for Chagas disease. Most of them are based on the development of host (human) antibodies directed against infection by parasites, although direct microscopic examination of blood smears can reveal the parasites themselves.
Unfortunately, if Chagas disease is not diagnosed and treated early, this infection progresses into the chronic phase. It is diagnosed at a later stage, when the disease is much more difficult to treat, because the damage to the internal organs that has developed with this disease is usually irreversible.
Another reason for this is that people who are infected are usually very poor, have primitive living conditions, and do not have access to medical care.
If a person lives in an area where steps associated with Chagas disease are common, the acute phase is often not diagnosed. Most acute infections go undiagnosed because many people develop nonspecific symptoms.
Chronic inflammation develops as the body reacts to the parasites, how they affect the nerve cells or neurons in these tissues, causing changes in the electrical conduction of the heart (arrhythmias) and insufficient muscle tone in the intestines.
These symptoms are associated with damage to the organs, which is caused by the constant presence of astelin in the tissues of these organs.
Heart rhythm disorders Fast heartbeat Syncope (syncope) Cardiomyopathy (chronic disease of the heart muscle) Heart failure Shortness of azelastine (shortness of breath) Emphysema Seizures Sudden death Chronic abdominal pain Chronic constipation Dilated colon Difficulty swallowing.
The symptoms of chronic Chagas disease vary depending on which organs are most affected. In most cases, lesions of the heart and/or gastrointestinal tract show the most severe symptoms. Symptoms of chronic Chagas disease may include the following:
While HIV/AIDS slowly attacks the immune system, Chagas disease slowly attacks the heart and gastrointestinal tissues. Other researchers consider such a comparison to be unfounded advertising or a publicity stunt to popularize information about Chagas disease.
However, this stage may only last 10-20 years before some patients develop chronic symptoms, which occur in about 10%-30% of those infected.
Most researchers believe that the intermediate andwhether the indeterminate phase has no symptoms. This stage can last throughout a person's life, and they may not even be aware that they have Chagas disease, especially if the symptoms of the acute phase were mild or completely absent.
For most people who develop symptoms outside of the acute phase, they resolve on their own within three to eight weeks. Sometimes an acute infection provokes chronic symptoms (see below), especially if the patient's immune system is weakened.
The symptoms of Chagas disease can be quite variable, ranging from no symptoms at all to severe and distressing symptoms. The first symptoms as the disease enters the acute phase may include some of the following:
Bedbugs that become part of the living conditions of people and their pets (cats, dogs) transmit T. cruzi to them. When T. cruzi is transmitted from bedbugs to humans or animals, and from humans to bedbugs, the life cycle is called the resident cycle. T. cruzi has been reported to be transmitted to humans through blood transfusions, organ transplants, from mother to child through the placenta, by mouth, inhalation, and laboratory instruments if not properly sterilized. Fortunately, these forms of transmission are very rare.
The life cycle of T. cruzi is complex, with several stages of development, both in the host insect (triatomy bug) and in mammalian (human and animal) recipients.